Wen, H., Andrejka, L., Ashton, J., Karess, R., and Lipsick, J.S. 2008. Epigenetic regulation of gene expression by Drosophila Myb and E2F2-RBF via the Myb-MuvB/dREAM complex. Genes & Development 22: 601-14. [pdf]

Ko ER, Ko D, Chen C, Lipsick JS. 2008. A conserved acidic patch in the Myb domain is required for activation of an endogenous target gene and for chromatin binding. Molecular Cancer 7: 77.  [pdf]

Manak JR, Wen H, Van T, Andrejka L, Lipsick JS. 2007. Loss of Drosophila Myb interrupts the progression of chromosome condensation. Nature Cell Biology 9: 581-7. [pdf]

Wang DM, Sevcikova S, Wen H, Roberts S, Lipsick JS. 2007. v-Myb represses the transcription of Ets-2. Oncogene 26: 1238-44. [pdf]

Navratilova J, Horvath V, Kozubik A, Lojek A, Lipsick J, Smarda J. 2007. p53 arrests growth and induces differentiation of v-Myb-transformed monoblasts. Differentiation. 75: 592-604. [pdf]

Fu S, Ganter B, Lipsick JS. 2006.  Myb proteins inhibit fibroblast transformation by v-Rel. Molecular Cancer 5: 54. [pdf]

Davidson CJ, Tirouvanziam R, Herzenberg LA, Lipsick JS. 2005. Functional evolution of the vertebrate Myb gene family: B-Myb, but neither A-Myb nor c-Myb, complements Drosophila Myb in hemocytes. Genetics 169: 215-29. [pdf].

Lipsick, JS. 2004. synMuv Verite-- Myb Comes Into Focus. Genes & Development 18: 2837-44. [pdf]

Tirouvanziam R, Davidson CJ, Lipsick JS, Herzenberg LA. 2004. Fluorescence-activated cell sorting (FACS) of Drosophila hemocytes reveals important functional similarities to mammalian leukocytes.  Proc Natl Acad Sci U S A 101: 2912-7. [pdf]

Beall, EL, Manak, JR, Zhou, S, Bell M, Lipsick JS, Botchan MR.  2002. Role for a Drosophila Myb-containing protein complex in site-specific DNA replication. Nature 420: 833-7. [pdf]

Manak JR, Mitiku N, Lipsick JS.  2002.  Mutation of the Drosophila homologue of the Myb proto-oncogene causes genomic instability. Proc. Natl. Acad. Sci. USA 99: 7438-43. [pdf] 

Wang DM, Lipsick JS. 2002. Mutational analysis of the transcriptional activation domains of v-Myb. Oncogene 21: 1611-5. [pdf]

Ladendorff NE , Wu S, Lipsick, JS. 2001. BS69, an adenovirus E1A-associated protein binds to and inhibits the transcriptional activity of c-Myb. Oncogene, 20:125-32. [pdf]

Wang DM, Dubendorff JW, Woo CH, Lipsick JS.  1999. Functional analysis of carboxy-terminal deletion mutants of c-Myb. J. Virol. 73: 5875-86. [pdf]

Lipsick JS, Wang DM. 1999. Transformation by v-Myb. Oncogene 18: 3047-55. [pdf]

Dubendorff, JW, Lipsick JS. 1999. Transcriptional regulation by the carboxyl-terminus of c-Myb depends upon both the Myb DNA-binding domain and the DNA recognition site. Oncogene 18: 3452-60. [pdf]

Ganter B, Lipsick JS. 1999. Myb and oncogenesis. Adv. Cancer Res. 76: 21-60. [pdf]

Ganter B, Chao ST , Lipsick JS. 1999. Transcriptional activation by the myb proteins requires a specific local promoter structure FEBS Lett. 460:401-10. [pdf]

Woo CH, Sopchak L, Lipsick JS. 1988. Overexpression of an alternatively spliced form of c-Myb results in increased transactivation and transforms avian myelomonoblasts. J. Virol. 72: 6813-21. [pdf]

Ohi R, Feoktistova A, McCann S, Valentine V, Look T, Lipsick JS, Gould KL. 1998. Myb-related Schizosaccharomyces pombe cdc5p is structurally and functionally conserved in eukaryotes. Mol. Cell. Biol. 18: 4097-108. [pdf]

Ganter B, Fu SL, Lipsick JS. 1998. D-type cyclins repress transcriptional activation by the v-Myb but not the c-Myb DNA-binding domain. EMBO Journal 17: 255-68. [pdf]

Ganter B, Lipsick JS. 1997. Myb binding sites within the N-ras promoter repress transcription. Oncogene 15:193-202. [pdf]

Engelke U, Wang DM, Lipsick JS. 1997. Cells transformed by a v-Myb-estrogen receptor fusion differentiate into multinucleated giant cells. J. Virol. 71:3760-6. [pdf]

Fu SL, Lipsick JS. 1997. Constitutive expression of full-length c-Myb transforms avian cells characteristic of both the monocytic and granulocytic lineages. Cell Growth Diff. 8:35-45. [pdf]

Zheng Z, Brockel C, Ganter B, Lipsick J, Sasaki M, Ogata K, Nishimura Y, Jardetzky O. 1996. Amide proton exchange rates in c-Myb DNA-binding domain. Quart. Magn. Res. in Biol. Med. 3:33-45.

Lipsick JS. 1996. One billion years of Myb. Oncogene 13:223-35.

Fu SL, Lipsick JS. 1996. FAETL motif required for leukemic transformation by v-Myb. J. Virol. 70:5600-10. [pdf]

Chen R, Fields S, Lipsick JS. 1995. Dissociation of transcriptional activation and oncogenic transformation by v-Myb. Oncogene 11:1771-9.

Dini PW, Eltman JT, Lipsick JS. 1995. Mutations in the DNA-binding and transcriptional activation domains of v-Myb cooperate in transformation. J. Virol. 69:2515-24. [pdf]

Engelke U, Smarda J, Lipsick JS. 1995. By-pass of TPA-induced differentiation and cell cycle arrest by the c-Myb DNA-binding domain. Oncogene 11:735-41.

Engelke U, Whittaker L, Lipsick JS. 1995. Weak transcriptional activation is sufficient for transformation by v-Myb. Virology 208:467-77. [pdf]

Smarda J, Sugarman J, Glass C, Lipsick JS. 1995. Retinoic acid receptor-a suppresses transformation by v-myb. Mol. Cell. Biol. 15:2474-81. [pdf]

Engelke U, Lipsick JS. 1994. Transformation of myelomonocytic cells by the avian myeloblastosis virus is determined by the v-myb oncogene, not by the unique long terminal repeats of the virus. J. Virol. 68:2752-55.

Smarda J, Lipsick JS. 1994. c-Myb prevents TPA-induced differentiation and cell death in v-Myb transformed monoblasts. Oncogene 9:237-45.

Aziz, N., J. Wu, J. W. Dubendorff, J. S. Lipsick, T. W. Sturgill, and T. P. Bender. 1993. c-Myb and v-Myb are differentially phosphorylated by p42mapk in vitro. Oncogene 8:2259-65.

Bin, X. and J. S. Lipsick. 1993. Individual repeats of Drosophila Myb can function in transformation by v-Myb. J. Virol. 67:7332-9.

Chen, R. H. and J. S. Lipsick. 1993. Differential transcriptional activation by v-myb and c-myb in animal cells and Saccharomyces cerevisiae. Mol. Cell. Biol. 13:4423-31.

Dini, P. W. and J. S. Lipsick. 1993. Oncogenic truncation of the first repeat of c-Myb decreases DNA-binding in vitro and in vivo. Mol. Cell. Biol. 13:7334-48.

Smarda, J. and J. S. Lipsick. 1993. Dicistronic selection for nuclear proteins in living animal cells. Gene 137:145-9.

Dubendorff, J. W., L. Whittaker, J. Eltman, and J. S. Lipsick. 1992. Carboxy-terminal elements of C-Myb negatively regulate transcriptional activation in cis and in trans. Genes Dev. 6:2524-35.

Garrido, C., D. Leprince, J. S. Lipsick, D. Stehelin, D. Gospodarowicz, and S. Saule. 1992. Definition of functional domains in P135gag-myb-ets and p48v-myb proteins required to maintain the response of neuroretina cells to basic fibroblast growth factor. J. Virol. 66:160-6.

Garrido, C., F. Gr:asser, J. S. Lipsick, D. Stehelin, and S. Saule. 1992. Protein truncation is not required for c-myb proto-oncogene activity in neuroretina cells. J. Virol. 66:6773-6.

Grasser, F. A., K. LaMontagne, L. Whittaker, S. Stohr, and J. S. Lipsick. 1992. A highly conserved cysteine in the v-Myb DNA-binding domain is essential for transformation and transcriptional trans-activation. Oncogene 7:1005-9.

Siu, G., A. L. Wurster, J. S. Lipsick, and S. M. Hedrick. 1992. Expression of the CD4 gene requires a Myb transcription factor. Mol. Cell. Biol. 12:1592-604.

Stober-Grasser, U., B. Brydolf, X. Bin, F. A. Grasser, R. A. Firtel, and J. S. Lipsick. 1992. The Myb DNA-binding domain is highly conserved in Dictyostelium discoideum. Oncogene 7:589-96.

Garcia, A., K. LaMontagne, D. Reavis, U. Stober-Grasser, and J. S. Lipsick. 1991. Determinants of sequence-specific DNA-binding by p48v-myb. Oncogene 6:265-73.

Grasser, F. A., T. Graf, and J. S. Lipsick. 1991. Protein truncation is required for the activation of the c-myb proto-oncogene. Mol. Cell. Biol. 11:3987-96.

Ibanez, C. E. and J. S. Lipsick. 1990. trans activation of gene expression by v-myb. Mol. Cell. Biol. 10:2285-93.

Lane, T. N., C. E. Ibanez, A. Garcia, T. Graf, and J. S. Lipsick. 1990. Transformation by v-myb correlates with transactivation of gene expression. Mol. Cell. Biol. 10:2591-8.

Ibanez, C. E., A. Garcia, U. Stober-Grasser, and J. S. Lipsick. 1988. DNA-binding activity associated with the v-myb oncogene product is not sufficient for transformation. J. Virol. 62:4398-402.

Ibanez, C. E. and J. S. Lipsick. 1988. Structural and functional domains of the myb oncogene: Requirements for nuclear transport, myeloid transformation, and colony formation. J. Virol. 62:1981-8.

Stober-Grasser, U. and J. S. Lipsick. 1988. Specific amino acid substitutions are not required for transformation by v-myb of avian myeloblastosis virus. J. Virol. 62:1093-6.

Lipsick, J. S. and C. E. Ibanez. 1987. env-encoded residues are not required for transformation by p48v-myb. J. Virol. 61:933-6.

Lipsick, J. S. 1987. v-myb does not prevent the expression of c-myb in avian erythroblasts. J. Virol. 61:3284-7.

Lipsick, J. S. and W. J. Boyle. 1987. c-myb protein expression is a late event during T-lymphocyte activation. Mol. Cell. Biol. 7:3358-60.

Boyle, W. J., J. S. Lipsick, and M. A. Baluda. 1986. Antibodies to the evolutionarily conserved aminoterminal region of the v-myb-encoded protein detect the c-myb protein in widely divergent metazoan species. Proc. Nat. Acad. Sci. USA 83:4685-9.

Lipsick, J. S., C. E. Ibanez, and M. A. Baluda. 1986. Expression of molecular clones of v-myb in avian and mammalian cells independently of transformation. J. Virol. 59:267-75.

Perbal, B., J. S. Lipsick, J. Svoboda, R. F. Silva, and M. A. Baluda. 1985. Biologically active proviral clone of myeloblastosis-associated virus type 1: Implications for the genesis of avian myeloblastosis virus. J. Virol. 56:240-4.

Boyle, W. J., M. A. Lampert, J. S. Lipsick, and M. A. Baluda. 1984. Avian myeloblastosis virus and E26 virus oncogene products are nuclear proteins. Proc. Nat. Acad. Sci. USA 81:4265-9.

Boyle, W. J., J. S. Lipsick, E. P. Reddy, and M. A. Baluda. 1983. Identification of the leukemogenic protein of avian myeloblastosis virus and of its normal cellular homologue. Proc. Nat. Acad. Sci. USA 80:2834-28.

Lipsick, J. 1974. On the Wire (poem). Atlantic Monthly 234:61.

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